A Review Of PEG400
GPR35 agonists (YE120, zaprinast, and pamoic acid) promoted wound repair in a concentration-dependent way independently of cell proliferation, While a selected GPR35 antagonist CID2745687, forskolin, and pertussis toxin reversed the YE120-induced effect. YE120 amplified the mRNA expression of fibronectin and its receptor integrin αfive, and ERK1/t